New CH.com Forum | |
http://www.clusterheadaches.com/cgi-bin/yabb2/YaBB.pl
Cluster Headache Help and Support >> Medications, Treatments, Therapies >> sitting up while sleeping http://www.clusterheadaches.com/cgi-bin/yabb2/YaBB.pl?num=1406037683 Message started by cc45713 on Jul 22nd, 2014 at 10:01am |
Title: sitting up while sleeping Post by cc45713 on Jul 22nd, 2014 at 10:01am
It could just be me but I think that laying down flat on the bed when I sleep triggers an attack. And sitting up gets me longer intervals of sleep time. Also, going to bed too late or staying up too long after an episode seems to trigger more.
I think there is something to sleep patterns and frequency and strength of attacks. |
Title: Re: sitting up while sleeping Post by Mike NZ on Jul 22nd, 2014 at 7:12pm
For people who get CH overnight I've seen quite a few post that sleeping in a position that isn't quite horizontal can help, so this could be in a reclining chair or something similar. However all you can do is to experiment and see what works for you.
|
Title: Re: sitting up while sleeping Post by cc45713 on Jul 22nd, 2014 at 8:03pm
Batch. You are a fountain of information. Thank you.
In addition to clusterheadaches.com have you ever considered a blog or website that can be accessed without digging through hundreds of posts? Because in all my years I've not run across anyone with as deep an understanding of clusters as you. |
Title: Re: sitting up while sleeping Post by Isto on Aug 1st, 2014 at 1:17am
Hello,
Please make sure that you are not suffering sleep aphnea ( = too little oxygen while sleeping). It might be good to make heavy execise when jumping out from bed to get oxygen level of blood high (atleast it worked with me before I started using acetium). Brg Isto |
Title: Re: sitting up while sleeping Post by BobG on Aug 1st, 2014 at 10:34pm
99% of my attacks happen between midnight and 2am. As soon as it wakes me I'm up, down an energy drink and pace until the pain ends. I learned many years ago not to go back to bed. Laying flat in bed will only trigger another attack. I have a very soft, comfortable (read expensive) La-Z-Boy just to finish my cluster night sitting up. In the last 3 and a half years I've probably clocked more hours in the chair than in bed.
|
Title: Re: sitting up while sleeping Post by Jovan on Aug 3rd, 2014 at 3:28am
I find sitting up in bed definitley helps. Also lying on my back with an extra pillow. Sleeping on my side will almost always trigger a CH.
|
Title: Re: sitting up while sleeping Post by didgens on Aug 12th, 2014 at 1:01pm
YES ,, YES YES YES ,,, my son told me that he started to feel a CH coming on on Saturday night ,, and he stood up and it stopped. this pushes me even harder wondering if it isn't acid reflux .. (you know the commercials where someone has to tilt the bed to sleep).. He also has such horrible heartburn during the cluster phase...
|
Title: Re: sitting up while sleeping Post by didgens on Aug 12th, 2014 at 1:02pm
also note. . the last 2 nights I make sure he take a tablespoon of Mylanta before going to sleep. no CH
|
Title: Re: sitting up while sleeping Post by Mike NZ on Aug 12th, 2014 at 4:03pm
The current research is showing that CH originates in the hypothalamus area of the brain.
Standing up will alter blood pressure, which could impact it. |
Title: Re: sitting up while sleeping Post by didgens on Aug 12th, 2014 at 4:32pm
not sure im sold on the hypothalamus theory. some posters on other sites seem to have gotten relief from resolving stomach issues. that would and has lead me in the Vagus nerve direction.
|
Title: Re: sitting up while sleeping Post by didgens on Aug 12th, 2014 at 4:35pm |
Title: Re: sitting up while sleeping Post by didgens on Aug 13th, 2014 at 6:13pm
The article you link to only states that there are areas of the brain that show activity because of the pain of the cluster headache. There is nothing in this article which specifically states that the CH originates in the brain itself.
Note that the headache was induced. Not observed as it occurs naturally which is the real key here. WHY does it occur, not what is reacting to it in our brains after onset. see excerpt below Discussion We observed areas of activation in acute cluster headache that fall into two broad groups: areas known to be involved in pain processing or response to pain, such as cingulate and insula cortex and thalamus; and areas activated specifically in cluster headache but not in other causes of head pain, notably the hypothalamic grey areas. These data suggest that primary headache syndromes share some processing pathways but equally can be distinguished on a functional neuroanatomical basis by areas of activation specific to the clinical presentation. Studies with PET have repeatedly given results that show activation of the anterior cingulate cortex on the sensation of somatic or visceral pain that are attributed to the emotional response to pain.13, 16 and 17 Activations in the insula have been shown after application of heat,16 and 18 subacutaneous injections of ethanol,19 somatosensory stimulation,20 and during cluster headache.13 Given its anatomical connections, the insula has been suggested as a relay of sensory information into the limbic system and is known to play an important part in the regulation of autonomic responses.21 Painful stimuli are significantly effective in activating the anterior insula, a region closely associated with both somatosensory and limbic systems. Such connections may provide one route through which nociceptive input is integrated with memory to allow full appreciation of the meaning and dangers of painful stimuli. In the acute pain state the thalamus is a site where activations would most be expected. Activation of the contralateral thalamus as a result of pain is known from studies on animals22 and functional imaging studies in human beings.16 and 17 The acute pain in cluster headache, induced activation bilaterally in the cerebellar hemispheres and in the vermis. There seems to be no direct nociceptive input to the cerebellum,23 and there is no clinical evidence that cerebellar lesions or stimulation affect pain sensation in human beings.16 However, there are some PET studies that report an activation in this area during experimental pain.16 and 24 In contrast to migraine,25 no brain stem activation was found during the acute attack compared with the resting state. This finding is remarkable because migraine and cluster headache are often discussed as associated disorders and similar compounds, such as ergotamine and sumatriptan, are used in the acute treatment of both types of headache. These data suggest that while primary headaches, such as migraine and cluster headache, may share a common pain pathway (the trigeminovascular innervation), the underlying pathogenesis differs substantially as might be inferred from the different patterns of presentation and responses to preventive agents.26 Substantial activations ascribable to cluster headache were observed in the ipsilateral hypothalamic grey area when compared with the headache-free state. Just as it is striking that no brain-stem activation occurs, which is in contrast to acute migraine,25 we have seen no hypothalamic activation in experimental pain induced by capsaicin injection into the forehead.27 Injection into the forehead would activate first division (ophthalmic) afferents which traverse the trigeminal division responsible for pain activation in cluster headache. Thus two other types of first division trigeminal nerve pain, while sharing neuroanatomical pathways with cluster headache, do not give rise to hypothalamic activation. Moreover, in the eight control patients who did not experience a headache after taking nitroglycerin, rCBF in the region of the hypothalamic grey area was not increased. This finding implies that the activation we have observed is involved in the pain process in a permissive or triggering manner rather than simply as a response to first division nociception per se. Hypothalamic activation in traumatic nociception has been observed in the hypothalamus proper and is a different more rostral area than we report.19 Moreover, Hsieh and colleagues19 report changes contralateral to the pain, whereas we report changes that are ipsilateral and in the hypothalamic grey area in the region of the circadian pacemaker neurons which is, therefore, an anatomically distinct area on the opposite side of the brain. Given that this area is involved in circadian rhythm and sleep-wake cycling, our data establish an involvement of this area of the hypothalamus as a primum movens in the acute cluster attack. Cluster headache has been attributed to an inflammatory process in the cavernous sinus and tributary veins.28 Inflammation has been thought to obliterate venous outflow from the cavernous sinus on one side, thus injuring the traversing sympathetic fibres of the intracranial internal carotid artery and its branches. According to this theory, the active period ends when the inflammation is suppressed and the sympathetic fibres partially or fully recover. This theory is based on abnormal findings with orbital phlebography in patients with cluster headache,29 and the fact that nitroglycerin and other vasodilators can induce a cluster attack.5 However, given the circadian rhythmicity and unilaterality of the symptoms, a purely vasogenic cause cannot explain the entire picture of cluster headache.30 Moreover, the frequency and pattern of pathological findings at orbital phlebography in cervicogenic headache, migraine, and tension-type headache is similar to that in cluster headache.31 Given that we have found an increased signal in the region of the cavernous sinus in the patients with acute cluster headache in this study and after capsaicin injection to the forehead in another PET study,27 it seems likely that the vascular changes are an epiphenomenon of activation of the trigeminovascular system.32 A radical reappraisal of the pathophysiology of cluster headache is needed. Our data establish that cluster headache, far from being a primarily vascular disorder, is a condition the genesis of which is to be found in the central nervous system in pacemaker or circadian regions of the hypothalamic grey matter. Further, we suggest that both cluster headache and migraine might usefully be regarded are neurovascular headaches to include the neural contribution to these important clinical syndromes. |
Title: Re: sitting up while sleeping Post by didgens on Aug 13th, 2014 at 6:14pm
also
Cluster headache has been attributed to an inflammatory process in the cavernous sinus and tributary veins. from above text |
Title: Re: sitting up while sleeping Post by didgens on Aug 13th, 2014 at 6:17pm
and as DUMB as it may sound to everyone ,, we are on Day 3 now ,, no stomach problems .. NO headaches. it may be his Cluster phase is over for now.. I do not know.. as it is all changing .. but in treating his stomach symptoms for the last 3 days ,, his head has improved.
|
Title: Re: sitting up while sleeping Post by didgens on Aug 14th, 2014 at 12:13pm
I would also like to site your study.. see the statement below
Acute cluster headache was provoked by inhalation of nitroglycerin (1·0–1·2 mg). so if the inhalation of nitro into the sinus cavity can induce cluster headache .. why cant acid reflux getting into the sinus cavity upon sleep or lying down do the same thing ? |
Title: Re: sitting up while sleeping Post by didgens on Aug 14th, 2014 at 5:14pm
Thanks Feisty ,, about 3-4 days in now with my son taking Mylanta .. he says he feels good ?? I dunno. Obviously something sprayed/inhaled in the sinus cavitiy bringing on a cluster headache as stated in the study makes me wonder what gets in the sinus cavitiy of "natural occurring" CH's to cause the problem.
|
Title: Re: sitting up while sleeping Post by didgens on Aug 14th, 2014 at 5:17pm |
Title: Re: sitting up while sleeping Post by didgens on Aug 14th, 2014 at 5:22pm
this article states that in the 90's researchers postulated that reflux material could also reach the naso-phrangeal and nasel district causing acute and and chronic rhino-sinusitis
|
Title: Re: sitting up while sleeping Post by didgens on Aug 14th, 2014 at 5:28pm |
New CH.com Forum » Powered by YaBB 2.4! YaBB © 2000-2009. All Rights Reserved. |